Fibrosis is a highly conserved and co-ordinated protective response to tissue injury. The interaction of multiple pathways, molecules and systems determines whether fibrosis is self-limiting and ...

Qiang (John) Ding is a Professor at the University of Alabama. His research Interests include fibrosis, tissue injury, tissue repair and remodeling, cognition, and cancers.

The excessive activity of repair cells in the early stages of tissue recovery sets the stage for fibrosis by priming the activation of an important growth factor, according to a new study.

Researchers at National Jewish Health and colleagues have completed the first study comparing lung macrophages in multiple ...

Idiopathic pulmonary fibrosis (IPF) is a progressive and fatal lung disease with limited treatment options. IPF is thought to result from repeated microinjuries to aging lung tissue, triggering ...

Lastly, reprogramming p16 Ink4a+ ECs through Kdr overexpression in a liver injury model significantly reduced fibrosis, ...

B7-33 is a synthetic peptide derived from the relaxin protein family, specifically designed to interact with the relaxin ...

A team of scientists at McMaster University in Ontario have created a bio-ink that can 3D print “breathable” lung tissue.

Scientists have now, for the first time, gained detailed insights into the dynamic remodeling of the tissue during lung repair.

Researchers characterize important regulators of tissue inflammation, fibrosis and regeneration by Boston University School of Medicine Credit: CC0 Public Domain ...

So, TREM2 in liver macrophages is critical for promoting tissue repair and preventing excessive fibrosis, suggesting its potential as a therapeutic target.